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    Fracture

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    1 : FRACTURE Presentation by, Ms.Vijayalakshmi.J M.Sc (N) Lecturer, RASS Academy CON
    2 : ANATOMY
    3 : ANATOMY………………….
    4 : BONE CELLS
    5 : DEFINITION A fracture is a complete or incomplete break in a bone resulting from the application of excessive force.
    6 : CLASSIFICATION OPEN (OR) CLOSED BY FRACTURE PATTERN LINEAR FRACTURE OBLIQUE FRACTURE LONGITUDINAL FRACTURE TRANSVERSE FRACTURE SPIRAL FRACTURE
    7 : BY TYPE AVULSION FRACTURE COMPRESSION FRACTURE COMMINUTED FRACTURE GREENSTICK FRACTURE IMPACTED FRACTURE PATHOLOGIC STRESS BY EPONYM COLLE’S FRACTURE POTT’S FRACTURE
    8 : BY APPEARANCE BURST FRACTURE CHIP FRACTURE COMPLETE FRACTURE DISPLACED FRACTURE BY POINT OF REFERENCE PROXIMAL MID SHAFT DISTAL BY ANATOMIC LOCATION
    9 : CLASSIFICATION………………
    10 : CLASSIFICATION…………………
    11 : OPEN FRACTURE
    12 :
    13 : CLOSED FRACTURE
    14 : AVULSION FRACTURE
    15 : COMMINUTED FRACTURE
    16 : COMPOUND FRACTURE
    17 :
    18 : GREEN STICK FRACTURE
    19 :
    20 : IMPACTED FRACTURE
    21 : STRESS,PATHOLOGIC
    22 : COMPRESSION FRACTURE
    23 : COLLE’S
    24 : POTT’S
    25 : COMPLETE FRACTURE
    26 : BURST FRACTURE
    27 : DISPLACED FRACTURE
    28 : CHIPS FRACTURE
    29 : FACTORS LEADING TO FRACTURE TRAUMA GENERAL SYSTEMIC DISORDERS (OSTEO GENESIS IMPERFECTA, RICKETS, OSTEOMALACIA, OSTEOPOROSIS, NEOPLASMS LIKE MULTIPLE MYELOMA, FIBROSARCOMA. OSTEOGENIC CARCINOMA, HEMANGIOMA.
    30 : PREDISPOSING FACTORS….. AGE. EXTRINSIC FACTORS (MAGNITUDE, DURATION, DIRECTION OF FORCE). INTRINSIC FACTORS (PROPERTIES OF BONE, ELASTICITY,FATIGUE STRENGTH, AND DENSITY). BEHAVIORAL FACTORS (SMOKING).
    31 : MECHANISM OF INJURY STRESS, DIRECT FORCE TAPPING, CRUSH SOFT TISSUE DAMAGE INFLAMMATORY RESPONSE FATIGUE OCCUR FRACTURE
    32 : CLINICAL MANIFESTATIONS LOCALIZED PAIN EDEMA AND SWELLING PAIN AND TENDERNESS MUSCLE SPASM DEFORMITY ECCHYMOSIS LOSS OF FUNCTION CREPITATION
    33 : FRACTURE HEALING
    34 : FACTURE HEALING FRACTURE HEMATOMA: WHEN FRACTURE OCCUS BLEEDING AND EDEMA CREATES HEMATOMA WHICH SURROUNDS ENTIRE FRAGMENTS. OCCURS 72 HOURS ATER INJURY.
    35 : GRANULATION TISSUE GRANULATION TISSUE: ACTIVE PHAGOCYTOSIS ABSORBSTHE PRODUCTS OF NECROSIS. THE HEMATOMA CONSISTS OF NEW BLOOD VESSELS, FIBROBLASTS AND OSTEOCLASTS. OCCURS 3 TO 14 DAYS.
    36 : FRACTURE HEALING CALLUS FORMATION: MINERLS ARE DEPOSITED AND UNORGANIZED NETWORK OF BONE IS FORMED, COMPOSED OF CARTILAGE, OSTEOBLASTS, CALCIUM AND PHOSPHORUS. BEGINS BY THE END OF 2ND WEEK.
    37 : FRACTURE HEALING OSSIFICATION: OCCURS FROM 3 WEEKS TO 6 MONTHS. THS WILL PREVENT MOVEMENTS AT THE FRACTURE SITE WHEN THE BONE IS STRESSED.
    38 : CONSOLIDATION: AS CALLUS CONTINUES TO DEVELOP THE DISTANCE BETWEEN BONE FRAGMENTS DIMINISHES AND EVENTUALLY CLOSES.
    39 : REMODELING: EXCES BONE TISSUE IS REABSORBED IN THE FINAL STAGEAND UNION IS COMPLETED. OCCURS UPTO A YEAR FOLLOWING INJURY.
    40 : MANAGEMENT FRACTURE REDUCTION TRACTION FRACRURE IMMOBILISATION CASTS INTERNAL FIXATION EXTERNAL FIXATION
    41 : OPEN REDUCTON
    42 : INTERNAL FIXATION
    43 : CLOSED REDUCTION
    44 : EXTERNAL FIXATORS
    45 : TRACTION
    46 : SKELETAL TRACTION
    47 : CASTS
    48 : COMPLICATIONS INFECTION COMPARTMENT SYNDROME (PARASTHESIA, PAIN, PRESSURE, PALLOR, PARLYSI PULSELESSNESS) VENOUS THROMBOSIS FAT EMBOLISM
    49 : COMPARTMENT SYNDROME
    50 : THANK YOU

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